Jumper’s Knee (patellar tendinitis) is an injury affecting the tendon connecting the kneecap (patella) to the shinbone (tibia). The patellar tendon helps the muscles extend the knee to kick a ball, run uphill, and jump up in the air.
Causes may include: overuse, repeated stress on the patellar tendon creating tiny tears in the tendon. When tears become numerous they cause pain from inflammation and a weakening of the tendon structure. This is most commonly seen in athletes whose sports involve frequent jumping, such as basketball and volleyball. This condition is the primary reason athletes retire from the sport.
Pain may include: pain in the section of the patellar tendon between the kneecap and the area where the tendon attaches to the shinbone, progressive pain in the area, pain when climbing up and downstairs, pain present as beginning a physical activity, or pain after an intense workout.
A.M.I.T. ® Explanation: This condition is caused by muscular imbalances of the quadriceps complex, gluteus maximus muscle group, and the gastrocnemius muscles. Historically, the quadriceps have been made up of four muscles: vastus medialis, vastus lateralis, vastus intermedius, and rectus femoris. We’ve discovered these muscles are actually divided into thirteen muscles: vastus medialis, upper, middle, lower, and oblique divisions, as is the vastus lateralis. The vastus intermedius is divided into two divisions, the medial and lateral divisions. The rectus femoris is divided into the straight head and reflected head. The last muscle is the small articularis genu. Each of these muscles has its own precise action. When these muscles become inhibited due to injury or overuse, their tendonous attachments become inflamed during the first six weeks following injury. After six weeks have lapsed, degenerative processes begin. The pain centers in the tendon/bone attachment, called the enthesis, generating intense pain to unload the muscle and protect it from tearing. Due to the muscle’s inability to stabilize the kneecap, it does not tract properly and the condylar surface becomes inflamed and more prone to degenerative changes.